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Publicação
Biological risks for neurological abnormalities associated with hyperbilirubinemia
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Orientador(es)
Resumo(s)
Unconjugated bilirubin (UCB) injury to glial cells leads to the secretion of glutamate and elicits a typical inflammatory response. Release of pro-inflammatory cytokines may influence gliogenesis and neurogenesis, and lead to deficits in learning and memo. - Fundacao para a Ciencia e a Tecnologia (FCT), Lisbon, Portugal. - The authors are grateful to the Fundacao para a Ciencia e a Tecnologia (FCT), Lisbon, Portugal, for their support.
Descrição
Palavras-chave
Obstetrics & Gynecology Pediatrics
Contexto Educativo
Citação
JOURNAL OF PERINATOLOGY. - Vol. 29, Suppl. Suppl. 1 (FEB 2009), p. S8-S13
Editora
NATURE PUBLISHING GROUP