Biological risks for neurological abnormalities associated with hyperbilirubinemia

Publicação

dc.contributor.author	Brites, D.
dc.contributor.author	Fernandes, A.
dc.contributor.author	Falcao, A. S.
dc.contributor.author	Gordo, A. C.
dc.contributor.author	Silva, R. F. M.
dc.contributor.author	Brito, M. A.
dc.date.accessioned	2015-12-30T10:18:20Z
dc.date.available	2015-12-30T10:18:20Z
dc.date.issued	2009
dc.description.abstract	Unconjugated bilirubin (UCB) injury to glial cells leads to the secretion of glutamate and elicits a typical inflammatory response. Release of pro-inflammatory cytokines may influence gliogenesis and neurogenesis, and lead to deficits in learning and memo. - Fundacao para a Ciencia e a Tecnologia (FCT), Lisbon, Portugal. - The authors are grateful to the Fundacao para a Ciencia e a Tecnologia (FCT), Lisbon, Portugal, for their support.
dc.format	application/pdf
dc.identifier.citation	JOURNAL OF PERINATOLOGY. - Vol. 29, Suppl. Suppl. 1 (FEB 2009), p. S8-S13
dc.identifier.doi	http://dx.doi.org/10.1038/jp.2008.214
dc.identifier.issn	0743-8346
dc.identifier.uri	http://hdl.handle.net/10451/21538
dc.language.iso	eng
dc.publisher	NATURE PUBLISHING GROUP
dc.subject	Obstetrics & Gynecology
dc.subject	Pediatrics
dc.title	Biological risks for neurological abnormalities associated with hyperbilirubinemia
dc.type	journal article
dspace.entity.type	Publication
oaire.citation.endPage	S13	por
oaire.citation.startPage	S8	por
oaire.citation.title	JOURNAL OF PERINATOLOGY	por
oaire.citation.volume	Vol. 29	por
rcaap.rights	restrictedAccess
rcaap.type	article

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