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Dipeptidyl‐peptidase IV inhibitor‐associated bullous pemphigoid efficiently treated with omalizumab

dc.contributor.authorGarrido, Pedro
dc.contributor.authorAlexandre, Maria Inês
dc.contributor.authorTravassos, Ana Rita
dc.contributor.authorFilipe, Paulo
dc.date.accessioned2021-09-30T11:46:51Z
dc.date.available2021-09-30T11:46:51Z
dc.date.issued2020
dc.description© 2020 Wiley Periodicals LLC.pt_PT
dc.description.abstractBullous pemphigoid (BP) is the most common autoimmune blistering disease. High potency topical and/or systemic corticosteroids as well as immunosuppressants are the current mainstay of treatment. However, long-term steroid therapy and systemic immunosuppression may result in significant morbidity and mortality. Contribution of IgE to BP pathogenesis remains under investigation, but increasing evidence supports a pathogenic significance. Omalizumab is a recombinant humanized monoclonal anti-IgE antibody that has recently emerged as a novel promising treatment alternative for BP.pt_PT
dc.description.versioninfo:eu-repo/semantics/publishedVersionpt_PT
dc.identifier.citationDermatol Ther. 2020 Nov;33(6):e14160pt_PT
dc.identifier.doi10.1111/dth.14160pt_PT
dc.identifier.eissn1529-8019
dc.identifier.issn1396-0296
dc.identifier.urihttp://hdl.handle.net/10451/49720
dc.language.isoengpt_PT
dc.peerreviewedyespt_PT
dc.publisherJohn Wiley & Sons, Inc.pt_PT
dc.relation.publisherversionhttps://onlinelibrary.wiley.com/journal/15298019pt_PT
dc.titleDipeptidyl‐peptidase IV inhibitor‐associated bullous pemphigoid efficiently treated with omalizumabpt_PT
dc.typejournal article
dspace.entity.typePublication
oaire.citation.issue6pt_PT
oaire.citation.titleDermatologic Therapypt_PT
oaire.citation.volume33pt_PT
person.familyNameGarrido
person.familyNameFilipe
person.givenNamePedro
person.givenNamePaulo
person.identifier.ciencia-id171A-309C-C397
person.identifier.orcid0000-0002-1048-3056
person.identifier.orcid0000-0002-7337-6493
person.identifier.scopus-author-id7003448874
rcaap.rightsrestrictedAccesspt_PT
rcaap.typearticlept_PT
relation.isAuthorOfPublicationb8bed940-e57b-4d35-a1b2-9ad490e78df5
relation.isAuthorOfPublicationac4119d7-4c3c-471a-9723-f8b8e7543f55
relation.isAuthorOfPublication.latestForDiscoveryb8bed940-e57b-4d35-a1b2-9ad490e78df5

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