Brites, D.Fernandes, A.Falcao, A. S.Gordo, A. C.Silva, R. F. M.Brito, M. A.2015-12-302015-12-302009JOURNAL OF PERINATOLOGY. - Vol. 29, Suppl. Suppl. 1 (FEB 2009), p. S8-S130743-8346http://hdl.handle.net/10451/21538Unconjugated bilirubin (UCB) injury to glial cells leads to the secretion of glutamate and elicits a typical inflammatory response. Release of pro-inflammatory cytokines may influence gliogenesis and neurogenesis, and lead to deficits in learning and memo. - Fundacao para a Ciencia e a Tecnologia (FCT), Lisbon, Portugal. - The authors are grateful to the Fundacao para a Ciencia e a Tecnologia (FCT), Lisbon, Portugal, for their support.application/pdfengObstetrics & GynecologyPediatricsBiological risks for neurological abnormalities associated with hyperbilirubinemiajournal articlehttp://dx.doi.org/10.1038/jp.2008.214