Differential inhibitory effect of long-chain acyl-CoA esters on succinate and glutamate transport into rat liver mitochondria and its possible implications for long-chain fatty acid oxidation defects

Ventura, FVRuiter, JIjlst, Lde Almeida, ITWanders, RJA2015-12-302015-12-302005MOLECULAR GENETICS AND METABOLISM. - Vol. 86, n. 3 (NOV 2005), p. 344-3521096-7192http://hdl.handle.net/10451/21636Long-chain fatty acid-oxidation defects are associated with a series of clinical and biochemical abnormalities, including accumulation of long-chain acyl-CoA esters which have been shown to inhibit several enzymes and transport systems that may disturb enapplication/pdfengBiochemistry & Molecular BiologyGenetics & HeredityMedicine, Research & ExperimentalDifferential inhibitory effect of long-chain acyl-CoA esters on succinate and glutamate transport into rat liver mitochondria and its possible implications for long-chain fatty acid oxidation defectsjournal articlehttp://dx.doi.org/10.1016/j.ymgme.2005.07.030