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Phenylketonuria as a protein misfolding disease

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dc.contributor.authorLeandro, J.
dc.contributor.authorSimonsen, N.
dc.contributor.authorde Almeida, Tavares,
dc.contributor.authorLeandro, P.
dc.contributor.authorFlatmark, T.
dc.date.accessioned2015-12-30T10:18:21Z
dc.date.available2015-12-30T10:18:21Z
dc.date.issued2008
dc.formatapplication/pdf
dc.identifier.citationJOURNAL OF INHERITED METABOLIC DISEASE. - Vol. 31, Suppl. Suppl. 1 (AUG 2008), p. 306
dc.identifier.issn0141-8955
dc.identifier.urihttp://hdl.handle.net/10451/21547
dc.language.isoeng
dc.publisherSPRINGER
dc.subjectEndocrinology & Metabolism
dc.subjectGenetics & Heredity
dc.titlePhenylketonuria as a protein misfolding disease
dc.titleMutant PG46S human phenylalanine hydroxylase has a propensity to self-associate and form amyloid fibrils
dc.typejournal article
dspace.entity.typePublication
oaire.citation.startPage306por
oaire.citation.titleJOURNAL OF INHERITED METABOLIC DISEASEpor
oaire.citation.volumeVol. 31por
rcaap.rightsrestrictedAccess
rcaap.typearticle

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